Histone H2B Induces Retinal Ganglion Cell Death Through Toll-like Receptor 4 in the Vitreous of Acute Primary Angle Closure Patients

Lab.Equipment makes sure that you get your regular Histopathology industry news fix whenever you want it. Check out today’s article first appearing on Laboratory Investigation:

Increased levels of histone H2B (H2B), a damage-associated protein, are found in the vitreous of patients with acute primary angle closure. Elevated H2B causes severe inflammation and subsequent retinal ganglion cell death through toll-like receptor 4 (TLR4) signaling. These results provide new insight for the mechanism of retinal ganglia degeneration.... Want to read more? Visit the Laboratory Investigation​ Blog.

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