mTORC2 Signaling is Required for Liver Regeneration in a Cholestatic Liver Injury Murine Model
May 06, 2020 | Histopathology
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Cholestatic liver injury may lead to a series of hepatobiliary syndromes, which can progress to cirrhosis and impaired liver regeneration, eventually resulting in liver-related death. It is known that the mammalian target of rapamycin complex 2 (mTORC2) is a major regulator of liver metabolism and tumor development. However, the role of mTORC2 signaling in cholestatic liver injury has not been characterized to date. In this study, we generated liver specific Rictor knockout mice to block the mTORC2 signaling pathway.... Want to read more? Visit the The American Journal of Pathology Blog.
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