Genetic Deletion of Galectin-3 Altered the Temporal Evolution of Macrophage Infiltration and Healing Affecting the Cardiac Remodeling and Function After Myocardial Infarction in Mice

June 01, 2020 | Histopathology

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We studied the role of Gal-3 in the expression of alternative activation markers (M2) on macrophage, cytokine expression and fibrosis through the temporal evolution of healing, ventricular remodeling and function after myocardial infarction (MI) in mice. Male C57BL/6J and Gal-3KO mice were subjected to permanent coronary artery ligation or sham for 1 and 4 weeks. We studied: 1) mortality; 2) macrophage infiltration and expression of markers of alternative activation at the infarct zone; 3) cytokine expression; 4) MMP-2 activity; 5) fibrosis; 6) cardiac function and remodeling... Read the complete article at The American Journal of Pathology Blog.

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